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;Feeding of MCT (coconut oil) to rats resulted in animals of low body weight, small fat deposits and excellent survival rate. This deserves emphasis because of the beneficial influence of low body weight on aging and arteriosclerosis. :http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=ShowDetailView&TermToSearch=3519928&ordinalpos=251&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum

;Coconut Oil Reduces Plasma Insulin and Glucose Concentrations - Weights of the total body and tissues did not differ significantly between the two groups of animals but plasma glucose and insulin concentrations were lower in the CO group. :http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=ShowDetailView&TermToSearch=10655979&ordinalpos=89&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum

;Coconut Oil Increases Sphingomyelin - Rats were fed on purified, high-cholesterol diets with either coconut fat or olive-oil (180 g/kg). During the course of the experiment, the concentration of plasma sphingomyelin rose in the coconut-fat group and remained constant in the olive-oil group. :http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=ShowDetailView&TermToSearch=10953679&ordinalpos=81&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum

;Lauric Acid is the primary component of coconut oil, and contains antimicrobial properties. :http://en.wikipedia.org/wiki/Lauric_acid

;Herpes virus and cytomegalovirus are inhibited by the antimicrobial lipid monolaurin which is only formed in the body if lauric acid is in the diet. :http://articles.mercola.com/sites/articles/archive/2001/07/28/coconut-health2.aspx

;Where fat comes from determines whether the body can metabolize it effectively. Researchers at Washington University School of Medicine in St. Louis have found that the "old" fat stored in the body's peripheral tissues -- that is, around the belly, thighs or bottom -- can't be burned efficiently unless "new" fat is eaten in the diet or made in the liver. [...] "When we took dietary fat away from the FASKOL (Fatty Acid Synthase KnockOut in Liver) mice, their livers quickly filled with fat," says senior investigator Clay F. Semenkovich, M.D., professor of medicine and of cell biology and physiology. "Their 'old' fat stores mobilized to the liver, but their livers could not initiate fat burning, and the fat just accumulated. We concluded that to regulate fat burning, the liver needs 'new' fat." :http://www.eurekalert.org/pub_releases/2005-05/wuso-nfi050905.php